{"id":67,"date":"2015-04-15T22:37:05","date_gmt":"2015-04-16T01:37:05","guid":{"rendered":"http:\/\/www.ufrgs.br\/patologiageral\/?p=67"},"modified":"2015-04-15T22:37:05","modified_gmt":"2015-04-16T01:37:05","slug":"degeneracao-hidropica","status":"publish","type":"post","link":"https:\/\/www.ufrgs.br\/patologiageral\/degeneracao-hidropica\/","title":{"rendered":"Degenera\u00e7\u00e3o Hidr\u00f3pica"},"content":{"rendered":"<p>&#8211; Degenera\u00e7\u00e3o hidr\u00f3pica (edema celular ou degenera\u00e7\u00e3o vacuolar) \u00e9 a primeira manifesta\u00e7\u00e3o de quase todas as formas de dano celular. (Robbins &amp; Cotran, 2015)<\/p>\n<p>&#8211; \u00c9 uma altera\u00e7\u00e3o revers\u00edvel n\u00e3o-letal.<\/p>\n<p>&#8211;\u00a0O ac\u00famulo intracelular de \u00e1gua ocorre pela incapacidade da c\u00e9lula de manter o equil\u00edbrio i\u00f4nico e a homeostasia de fluidos, em fun\u00e7\u00e3o de falha nas bombas dependentes de energia das membranas celulares.<\/p>\n<p>&#8211; Quando\u00a0ocorre em um \u00f3rg\u00e3o inteiro, este pode se apresentar p\u00e1lido, com aumento do peso e turgor.<\/p>\n<p>Exemplo:<\/p>\n<p>DEGENERA\u00c7\u00c3O HIDR\u00d3PICA EM PAR\u00caNQUIMA HEP\u00c1TICO<\/p>\n<figure id=\"attachment_98\" aria-describedby=\"caption-attachment-98\" style=\"width: 474px\" class=\"wp-caption aligncenter\"><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidr6.jpg\"><img loading=\"lazy\" class=\"wp-image-98 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidr6-1024x768.jpg\" alt=\"deg hidr6\" width=\"474\" height=\"356\" \/><\/a><figcaption id=\"caption-attachment-98\" class=\"wp-caption-text\">F\u00edgado, HE 10X.<\/figcaption><\/figure>\n<p>&nbsp;<\/p>\n<p>Observe\u00a0a seguir\u00a0detalhe da imagem anterior, onde se identificam zonas de hepat\u00f3citos claros com edema celular circundando a veia centrolobular, enquanto os hepat\u00f3citos que circundam o espa\u00e7o porta est\u00e3o mais eosinof\u00edlicos.<\/p>\n<figure id=\"attachment_70\" aria-describedby=\"caption-attachment-70\" style=\"width: 474px\" class=\"wp-caption aligncenter\"><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidropica2-e1429146646768.png\"><img loading=\"lazy\" class=\"wp-image-70 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidropica2-e1429146646768-1024x778.png\" alt=\"deg hidropica2\" width=\"474\" height=\"360\" srcset=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidropica2-e1429146646768-1024x778.png 1024w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidropica2-e1429146646768-300x228.png 300w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidropica2-e1429146646768.png 1238w\" sizes=\"(max-width: 474px) 100vw, 474px\" \/><\/a><figcaption id=\"caption-attachment-70\" class=\"wp-caption-text\">F\u00edgado, HE 10X.<\/figcaption><\/figure>\n<p>A seguir, em maior aumento, hepat\u00f3citos hidr\u00f3picos com citoplasma claro e\u00a0n\u00facleo central, circundando veia centrolobular.<\/p>\n<figure id=\"attachment_97\" aria-describedby=\"caption-attachment-97\" style=\"width: 474px\" class=\"wp-caption aligncenter\"><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidr5.jpg\"><img loading=\"lazy\" class=\"wp-image-97 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidr5-1024x768.jpg\" alt=\"deg hidr5\" width=\"474\" height=\"356\" \/><\/a><figcaption id=\"caption-attachment-97\" class=\"wp-caption-text\">F\u00edgado, HE 50X.<\/figcaption><\/figure>\n<p>&nbsp;<\/p>\n<p>Abaixo hepat\u00f3citos mais preservado\u00a0circundando espa\u00e7o-porta.<\/p>\n<figure id=\"attachment_96\" aria-describedby=\"caption-attachment-96\" style=\"width: 474px\" class=\"wp-caption aligncenter\"><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/de-hid3.jpg\"><img loading=\"lazy\" class=\"wp-image-96 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/de-hid3-1024x768.jpg\" alt=\"de hid3\" width=\"474\" height=\"356\" srcset=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/de-hid3-1024x768.jpg 1024w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/de-hid3-300x225.jpg 300w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/de-hid3.jpg 1600w\" sizes=\"(max-width: 474px) 100vw, 474px\" \/><\/a><figcaption id=\"caption-attachment-96\" class=\"wp-caption-text\">F\u00edgado, HE 50 X.<\/figcaption><\/figure>\n<p>&nbsp;<\/p>\n<p>A seguir, em maior aumento, detalhe do edema celular hepatocit\u00e1rio, caracterizado por clareamento citoplasm\u00e1tico, manuten\u00e7\u00e3o do n\u00facleo em uma posi\u00e7\u00e3o central e\u00a0distor\u00e7\u00e3o arquitetural dos cord\u00f5es hepatocit\u00e1rios com compress\u00e3o dos sinusoides.<\/p>\n<p><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hid2.jpg\"><img loading=\"lazy\" class=\"aligncenter wp-image-95 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hid2-1024x768.jpg\" alt=\"deg hid2\" width=\"474\" height=\"356\" srcset=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hid2-1024x768.jpg 1024w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hid2-300x225.jpg 300w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hid2.jpg 1600w\" sizes=\"(max-width: 474px) 100vw, 474px\" \/><\/a><\/p>\n<figure id=\"attachment_93\" aria-describedby=\"caption-attachment-93\" style=\"width: 474px\" class=\"wp-caption aligncenter\"><a href=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidro2.jpg\"><img loading=\"lazy\" class=\"wp-image-93 size-large\" src=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidro2-1024x768.jpg\" alt=\"deg hidro2\" width=\"474\" height=\"356\" srcset=\"https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidro2-1024x768.jpg 1024w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidro2-300x225.jpg 300w, https:\/\/www.ufrgs.br\/patologiageral\/wp-content\/uploads\/2015\/04\/deg-hidro2.jpg 1600w\" sizes=\"(max-width: 474px) 100vw, 474px\" \/><\/a><figcaption id=\"caption-attachment-93\" class=\"wp-caption-text\">F\u00edgado HE 400X.<\/figcaption><\/figure>\n<p>&nbsp;<\/p>\n<p><!--more--><\/p>\n<p>Leia mais:<\/p>\n<p><a title=\"http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2740913\/\" href=\"http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2740913\/\">http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC2740913\/<\/a><\/p>\n<p>&nbsp;<\/p>\n<p>&nbsp;<\/p>\n","protected":false},"excerpt":{"rendered":"<p>&#8211; Degenera\u00e7\u00e3o hidr\u00f3pica (edema celular ou degenera\u00e7\u00e3o vacuolar) \u00e9 a primeira manifesta\u00e7\u00e3o de quase todas as formas de dano celular. (Robbins &amp; Cotran, 2015) &#8211; \u00c9 uma altera\u00e7\u00e3o revers\u00edvel n\u00e3o-letal. &#8211;\u00a0O ac\u00famulo intracelular de \u00e1gua ocorre pela incapacidade da c\u00e9lula de manter o equil\u00edbrio i\u00f4nico e a homeostasia de fluidos, em fun\u00e7\u00e3o de falha nas &hellip; <a href=\"https:\/\/www.ufrgs.br\/patologiageral\/degeneracao-hidropica\/\" class=\"more-link\">Continue lendo <span class=\"screen-reader-text\">Degenera\u00e7\u00e3o Hidr\u00f3pica<\/span> <span class=\"meta-nav\">&rarr;<\/span><\/a><\/p>\n","protected":false},"author":1,"featured_media":0,"comment_status":"closed","ping_status":"open","sticky":false,"template":"","format":"standard","meta":[],"categories":[4],"tags":[],"_links":{"self":[{"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/posts\/67"}],"collection":[{"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/users\/1"}],"replies":[{"embeddable":true,"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/comments?post=67"}],"version-history":[{"count":0,"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/posts\/67\/revisions"}],"wp:attachment":[{"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/media?parent=67"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/categories?post=67"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/www.ufrgs.br\/patologiageral\/wp-json\/wp\/v2\/tags?post=67"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}